Current Focus of Research:
Proteins are naturally occurring polymers comprised
of amino acid subunits. The advantage of these molecules is their
ability to catalyze a wide range of complex chemistries and engage
in countless exquisitely specific molecular interactions, yet each
protein is itself constructed from a single synthetic chemistry and
its function is perfectly contained within a simple genetic code.
The ability of this class of molecule to perform highly complex tasks,
combined with their relative ease of synthesis by microorganisms,
has spurred the development of an industry pursuing the use of proteins
in separations, diagnostics, and numerous medical therapies.
Protein pharmaceuticals, also called biopharmaceuticals,
represent a rapidly developing product area in industry. In many
cases, the extreme specificity of molecular interations required
to inhibit certain disease states has limited the success of small
organic pharmaceuticals in treatment. Protein reagents have the
potential to overcome this obstacle; however, the utility of proteins
as therapeutic agents has been limited by the lack of a quantitative
engineering approach to developing proteins with the appropriate
molecular properties. My goal is to develop a program of research
addressing this need. This research must consider several critical
points:
1. Selection of a molecular scaffold with
properties suitable for the intended application (e.g, tissue penetration
properties, thermal stability).
2. Development of protein engineering processes
for quantitatively altering the functional properties of the chosen
reagent to optimize for the specific application.
3. Quantitative analysis of the relevant molecular
properties and combination of this knowledge with protein engineering
processes for molecular design.
A particular target application of this research is
engineering the immune response. Numerous devastating diseases result
from destruction of the body's own tissues due to attack by the cellular
immune system (autoimmunity). Misidentification of self in autoimmunity
and immunological attack of transplanted tissues, as well as failure
of the immune system to identify certain harmful entities (e.g., certain
viruses or tumor cells), is essentially a problem of molecular recognition.
Inhibiting undesirable immune responses or stimulating immune responses
against novel targets are problems most likely to be solved by taking
advantage of the unique properties of engineered protein reagents.
Research in this area makes use of tools from molecular biology, protein
chemistry, and biophysics.
Selected Publications (more info):
S. Subramanian, E.T. Boder, and D.E. Discher: Phylogenetic Divergence in Human SIRPa-CD47 Interactions Reveals Locus of Species-specificity: Implications for the Binding Site. J. Biol. Chem. (in press).
J.H. Lee, M. Goulian, and E.T. Boder: Autocatalytic Activation of Influenza Hemagglutinin. J. Mol. Biol., 364:275-282 (2006).
L.R. Pepper, D.A. Hammer, and E.T. Boder: Rolling Adhesion of aL I Domain Mutants Decorrelated from Binding Affinity J. Mol. Biol., 360:37-44 (2006).
S. Park, Y. Xu, X.F. Stowell, F. Gai, J.G. Saven, and E.T. Boder: Limitations of yeast surface display in engineering proteins of high thermostability. Prot. Eng. Des. Sel., 19:211-217 (2006).
P. Derr, E. Boder, and M. Goulian: Genetic selection for new bacterial chemoreceptors. J. Mol. Biol. 355:923-932 (2006).
S. Subramanian, R. Parthasarathy, E.T. Boder, and D.E. Discher: Species-specific adhesive interactions between CD47 and human SIRPa. Blood, 107:2548-2556 (2006).
R. Parthasarathy, S. Subramanian, E.T. Boder*, and D.E. Discher: Post-translational regulation of expression and conformation of an immunoglobulin domain in yeast surface display. Biotechnol. Bioeng., 93:159-168 (2006).
R. Parthasarathy, J. Bajaj, and E.T. Boder: Immobilized biotin ligase via surface display of E. coli BirA on Saccharomyces cerevisiae. Biotechnol. Prog., 21:1627-1631 (2005).
E.T. Boder, J.R. Bill, A.W. Nields, P.C. Marrack, and J.W. Kappler: Yeast surface display of a noncovalent MHC class II heterodimer complexed with antigenic peptide. Biotechnol. Bioeng., 92:485-491 (2005).
S. Park, E.T. Boder, and J.G. Saven: Modulating the DNA affinity of Elk-1 with computationally selected mutations. J. Mol. Biol., 348:75-83 (2005).
S.J. Park, H. Kono, W. Wang, E.T. Boder, and J.G. Saven: Progress in the development and application of computational methods for probabilistic protein design. Computers Chem. Eng., 29:407-21 (2005).
|